• ✺roguetrick✺@lemmy.world
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      2 days ago

      It would take a very large dose to affect the heart and even then it would just lead to a slower heart rate instead of stopping it. The heart does not need nerves to tell it to beat and it’s action potential triggering is different than muscles and nerves. They’ll be brain dead from being without oxygen before they’re heart dead, similar to opioid overdoses.

        • ggppjj@lemmy.world
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          2 days ago

          I would personally imagine that you may need to be defibrillated at some point but otherwise probably yes? The toxins are causing the paralysis and people do survive it so I can only imagine that the heart takes back over after a certain amount of effort. Otherwise, I don’t actually know.

          • RedditRefugee69@lemmynsfw.com
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            2 days ago

            Defibrillation is only useful if the problem is your heart is doing some kind of fibrillation.

            If it’s not beating at all, other methods like manual massage or chemical restarts (epinephrine) are the right move.

            • ggppjj@lemmy.world
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              2 days ago

              Gotcha. My CPR training was so long ago, and the only relevant information that really stuck with me was “the AED will directly instruct you if it thinks a shock is helpful based on what it detects”, after that the specifics just kinda fell through my brain.

          • ✺roguetrick✺@lemmy.world
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            2 days ago

            You might need external/transesophageal pacing with a severe exposure to TTX, but that would only be temporary. It shouldn’t cause v fib.